AnUser
#2
If you exercise on a bike hard enough you’ll have a lot of lactate surrounding the mitochondria, as a byproduct from oxygen being converted to pyruvate which enters the mitochondria.
If you have trained a lot at maximum fat oxidation at the point before the lactate surrounding the mitochondria increase so much to increase hydrogen and possibly lead to fatigue and stopping the activity… and upregulating lactate receptors over time to improve the ability to bring lactate into the mitochondria.
So you have a lot more MCT1 receptors, and can therefore the lactate outside the mitochondria is transported very quickly and does not accumulate outside. It will show up as a low resting lactate level and a much higher ability to produce force without lactate accumulating outside the mitochondria (which is a lot of lactate in blood stream).
Not only will apparently MCT1 receptors be upregulated… so will MPC, and all of the components in the krebs cycle (mitochondrial respiration) from exercise and optimally so in Inigo’s experience from training at maximum fat oxidation or before lactate starts accumulating a lot.
I produced a slide yesterday as part of a presentation on this that I thought people might like to see a copy of.
1 Like
