#21

I have incorporated zone 2 running in the last 2 weeks. It’s very different from how Ive trained in the past. Have you done this for long and felt any increase in recovery from harder training sessions?

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#22

Zone 2 has a few promised benefits. I’ve seen a few.
(1) less stress requires less recovery (easy for this to be true; saves muscles for a few hard sessions a week; polarized training)
(2) builds more power at the same, moderate HR (over time)
(3) increases fat burning (hard for me to measure)

It’s the way to go for me.

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#23

I think I’m becoming a fan. It won’t give me the rush and sense of feeling alive as more intense workouts. But it’s so easy. I never have to force myself, and I always feel refreshed afterwords, never exhausted.

Don’t know if this is good practice but it even feels safe to do with a mild cold. Symptoms have remained the same or felt better afterwards.
Honestly think that it just lubricates everything.
When cold is completely gone I will start ending every run with a few minutes in zone 5, and throw In one set of intervals each week.

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#24

Here’s the zone 2 man himself.

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#25

At one point I did 3-7 day fasts every month or two. With the purposes of building muscles and the initiation and extensions of autophagy I no longer fast those extended periods. Once I began taking Rapamycin 1 1/2 years ago, I recognized that I did not want to conflict with, or interrupt it’s mTOR autophagy actions. After taking my Rapamycin each Saturday morning, I fast that day and do not exercise. My fasting between meals during this Saturday every week accounts for approximately 40 hours. I don’t want to absorb any foods that could excite my mTOR with proteins. I believe this probably gives me at least a continuous 24 hours of autophagy each week while taking the rapamycin. Incidentally, I also fast those Saturdays when I am off of the Rapamycin. Any thoughts here are appreciated.

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#26

This idea is not bad but it’s based on what I think is an erroneous assumption that rapamycin will significantly lower mTOR when mTOR is already very low. We know that rapamycin even at massive concentrations doesn’t fully inhibit mTOR and we know that fasting by itself decreases mTOR and increases autophagy a lot. Therefore I don’t think there is a good basis to think rapamycin will lower mTOR a lot more when one is fasting. I would argue that the main point of taking rapamycin is not to try to suppress mTOR more when it’s already low but to interfere with otherwise normal mTOR activation such as that which occurs in response to food. Note that in the mouse life span studies, the mice were given rapamycin with food.

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