#185

Yeah, Clinton. Have you ever fought before? Clinton has speed and agility over heavy and slow Trump, Clinton would knock him out within 10 seconds. I don’t think Trump has the strength to lift his arms to protect his face.

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#186

yes lots of speed and agility indeed. check it out here, lol dude
Bill and Hillary Clinton were spotted on the beach Tuesday during their annual Hamptons getaway | Daily Mail Online

#187

Not sure what did you just prove with that link, it seems like Clinton can walk on sand (uneven ground) pretty well unlike this guy

he also uses two hands two drink, so he has hard time keep his arms up, so can’t keep his guard up
Why would you think he can fight?

you are clearly just politically motivated, remove the politics and you get…
Burger fueled fat guy that can barely move vs. vegan skinny guy who is quite agile

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#188

Guys, please let’s avoid politics here. Future comments on politics will be deleted. Or the topic will be closed.

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#189

I am simply pushing back on this propagated stereotype that vegans are weak and sickly, whereas obese carnivores are strong and healthy. I did not choose this asinine example.

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#190
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#191

Powerful and compassionate video.

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#192

A new open access paper:

A novel carnivorous diet reduces brain telomere length

Developmental conditions can profoundly influence adult survival or longevity. One established correlate of longevity is the length of telomeres—non-coding DNA regions that protect chromosomal ends. Telomere length in adulthood can be influenced by environmental conditions during development, such as nutrient restriction. Yet, we lack experimental studies of how adult telomere length is affected by a different form of nutritional variation: diet type. Here, we asked how diet-type variation during larval development affects telomere length in multiple post-metamorphic somatic tissues of the Mexican spadefoot (Spea multiplicata ), an anuran species whose larvae develop on two qualitatively distinct diets: an ancestral omnivorous diet of detritus or a more novel carnivorous diet of live shrimp. We found that larvae developing on the novel shrimp diet developed into post-metamorphic frogs with shorter telomeres in the brain—a structure that is particularly vulnerable to harmful effects of nutritional adversity, such as oxidative stress. Given known links between telomere length and neurological health outcomes, our study suggests that a dietary transition to carnivory might carry costs in terms of compromised neural integrity later in life. This work highlights the lasting impact of a developmental diet on somatic maintenance and health.

https://royalsocietypublishing.org/doi/10.1098/rsbl.2024.0593

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#193

Dare I put this up… ?
Sure… why the hell not… HAHAHA evolution makes us thinking … moving carnivores … not sedentary monkeys. I dont want to go backwards. Lol

Link: How Much Meat Do Our Closest Genetic Relatives—Great Apes—Eat? A Biologist Answers

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#194

image

image

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#195

My personal experience is that my lipids went back to the levels I had in my early 30’s after getting off the carnivore diet. I’m certainly very thankful for rapa healing my gut so I could tolerate other foods again. I watched as my lipid panel slowly got worse with each blood draw for close to four years on carnivore. I still eat eggs and low fat dairy most days and lean meat a few times per week but I’m just no longer eating a solely high fat animal foods diet. For me plants and the nutrients and fibers that come with them have been amazing.

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#196

For the vast majority of people lifestyle changes isn’t enough to prevent the progression of atherosclerosis by having a low enough apoB ~30 mg/dl(?), and other risk factors when soft or calcified plaque is present, but lower is better of course.

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#197

And, we all differ to some degree in blood type… genetics… phenotypes. Nature does this as a way to promote survival of a species.

I do think rapamycin can do some plaque clearing which might also cause the appearance of more lipids in the blood.

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#198

Is this something your gut tells you, or have you seen this somewhere? Only curious because I am chock full o’ plaque.

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#199

As we know not enough research on rapamycin. But some of that possible atheroscleurosis benefit comes form some mice studies on CVD.

Link: https://academic.oup.com/cardiovascres/article/120/Supplement_1/cvae088.204/7684113

Rapamycin, an immunosuppressant, has shown promise in reducing atherosclerotic plaque development and promoting plaque stability in animal models, potentially by suppressing macrophage proliferation, lipid accumulation, and inflammation.

Here’s a more detailed explanation:

  • Anti-Atherosclerotic Effects:

    • Rapamycin has been shown to decrease plaque burden and enhance plaque stability in animal models.
    • It can reduce the growth of vascular lesions and inflammation associated with atherosclerosis.
    • Rapamycin may work by suppressing macrophage proliferation, lipid accumulation, and inhibiting intraplaque neoangiogenesis.
    • It can also promote regulatory T cells and reduce the accumulation of T cells, ABCs, and macrophages in plaques.

  • Mechanisms of Action:

    • Rapamycin’s anti-atherosclerotic effects are thought to be mediated through anti-inflammatory, lipid-modulatory, apoptosis, autophagy, and anti-thrombotic effects.
    • It can induce autophagy in macrophages, inhibit cell proliferation, reduce the engulfing of oxLDL in macrophages, and prohibit the formation of foam cells.

  • Potential Therapeutic Value:

    • Rapamycin could be a potential therapeutic to halt cardiovascular disease (CVD) progression.
    • Studies have shown that rapamycin can reduce the incidence of coronary restenosis after stenting.
    • Rapamycin may attenuate inflammation and enhance the stability of atherosclerotic plaques, independent of serum lipid levels.

  • Rapamycin and Alzheimer’s Disease:

    • Some studies suggest that rapamycin can reduce amyloid plaque deposition in mouse models of Alzheimer’s disease.
    • However, other research indicates that rapamycin may increase Alzheimer’s-associated plaques in mice.
    • The effects of rapamycin on Alzheimer’s disease are complex and require further investigation
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#200

Oh yeah, I did once hear about the use with stents… but WOW, I had not heard any of this about plaque

Rapamycin, the gift that keeps on giving

Merry Christmas and bless us everyone!

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#201

Yes sir. It’s much, much improved but not optimal so I’m starting meds.:smiley:
I think the combination will get me to perfect.

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